I do not have full statistics, but people who smoke cigarettes are less prone for developing post-covid, according to the statistics I have.
My immediate thought back then was is that nicotine somehow plays the role of NAD (= B3 vitamin), thus fixing one of the core mechanisms of acquiring mitochondrial dysfunction after the covid (NAD deprivation).
Couldnt it just be a selection effect? People who smoke cigarettes are less neurotic. I would assume people with long-covid are on average more neutrotic, as people with high neuroticism is more likely to attribute sickness to long-covid?
My immediate thought back then was is that nicotine somehow plays the role of NAD (= B3 vitamin), thus fixing one of the core mechanisms of acquiring mitochondrial dysfunction after the covid (NAD deprivation).