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When we keep patients ‘dry’ in ICU we aim to keep their urine output at a level that indicates safe glomerular filtration rates.

ACE2 is expressed extensively in renal tissue, so the virus is transported there in the blood, infects cells, hijacks the cellular apparatus to replicate, and then explodes to cell to continue its march of death. So I think your speculation is a bit misplaced




Ah, thanks!




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