The colloquial use of “dopamine” is more metaphorical than scientific. Your brain and body use dopamine in many different pathways for many different reasons. For example, the tremors in Parkinson’s disease are related to the degradation of certain movement-related dopaminergic pathways in your brain.
When most people self-diagnose as “low dopamine” or refer to addictive behaviors as a “dopamine hit”, they’re not exactly referring to hard science. Yes, dopamine is involved in motivational reward and anticipatory behavior and dopamine is also key to the rewarding aspects of certain drugs, but the reward system is much more complicated than a single chemical messenger. For example, the opioid system plays an important role in reward as well.
In terms of treating addiction disorders, understanding the fine details of neurotransmitters and reward pathways in the brain isn’t as relevant to therapy as you might hope. The high level concepts and therapeutic methods for changing people’s behavior don’t really depend on understanding how dopamine or serotonin operate in the brain.
For example, with internet addiction it’s important to enable people to understand why they are spending so much time on the internet. You need to give them the tools to recognize their behavior, perform some introspection, and intentionally take action to make different choices about how they spend their time. Relating this back to dopamine or reward systems is only really useful as a tool to make these mental models stick with the patients. If they feel a sense of understanding, they’re more likely to feel a sense of control over their actions, and in turn more likely to take action to change that behavior.
We already know what happens when you don’t have enough dopamine (Parkinson’s disease - dopamine analogues are used to treat it) and too much dopamine (schizophrenia - dopamine blockers are used to treat it). Saying more is better is a major over simplification.
Dopamine is involved in a number of different neural structures that all have different effects. Not to mention there are several dopamine receptors subtypes that all have different actions.
If you're trying to debug a computer system, you won't get very far by trying to adjust the voltage of the circuitry, whether you do it overall or try to tune specific areas. Imagining there needs to be "more ones" or "more zeros" is pretty simplistic.
On the other hand, talking to it is not likely to help either, although you may work out some of your own issues.
When most people self-diagnose as “low dopamine” or refer to addictive behaviors as a “dopamine hit”, they’re not exactly referring to hard science. Yes, dopamine is involved in motivational reward and anticipatory behavior and dopamine is also key to the rewarding aspects of certain drugs, but the reward system is much more complicated than a single chemical messenger. For example, the opioid system plays an important role in reward as well.
In terms of treating addiction disorders, understanding the fine details of neurotransmitters and reward pathways in the brain isn’t as relevant to therapy as you might hope. The high level concepts and therapeutic methods for changing people’s behavior don’t really depend on understanding how dopamine or serotonin operate in the brain.
For example, with internet addiction it’s important to enable people to understand why they are spending so much time on the internet. You need to give them the tools to recognize their behavior, perform some introspection, and intentionally take action to make different choices about how they spend their time. Relating this back to dopamine or reward systems is only really useful as a tool to make these mental models stick with the patients. If they feel a sense of understanding, they’re more likely to feel a sense of control over their actions, and in turn more likely to take action to change that behavior.